Titlebook: Anxiety and Anxiolytic Drugs; Florian Holsboer,Andreas Str?hle Book 2005 Springer-Verlag Berlin Heidelberg 2005 Anxiety.Anxiolytic Drugs.N

SNEER

https://doi.org/10.1007/978-3-642-53210-8 to play a causal role in the etiology and symptomatology of anxiety disorders. Indeed, there is increasing evidence from basic science that chronic stress-induced perturbation of CRH and AVP neurocircuitries may contribute to abnormal neuronal communication in conditions of pathological anxiety. An

EXCEL

https://doi.org/10.1007/978-3-642-53210-8substance P (SP) and its cognate receptor neurokinin 1 (NK1R) seem to play a particularly important role in higher brain functions. They are expressed at high levels in the limbic system, which is the neural basis of emotional responses. Three different lines of evidence from physiological studies s

Preserve

https://doi.org/10.1007/978-3-642-53210-8ty disorder, and obsessive—compulsive disorder. Controlled family studies reveal that all of these anxiety subtypes are familial, and twin studies suggest that the familial aggregation is attributable in part to genetic factors. Panic disorder and, its spectrum have the strongest magnitude of famili

陳列

https://doi.org/10.1007/978-3-662-36767-4panic disorder, has been increasing steadily during the past several years. Although the picture is far from complete yet—partly due to the large number of serotonin (5-HT) receptors and the often-disparate effects of receptor agonists and antagonists in animal models of anxiety—SSRIs and the 5-HT.

DAMN

https://doi.org/10.1007/978-3-662-36767-4e systems have been demonstrated to play important roles in the behaviors associated with fear and anxiety-producing stimuli. Long-term dysregulation of these systems appears to contribute to the development of anxiety disorders, including panic disorder, posttraumatic stress disorder (PTSD), and so

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忍耐

蝕刻術(shù)

Zeit-Strom-Verhalten bis zum Unterbrechen,lasticity may play a role in the pathophysiology of anxiety. Intracellular signaling pathways are known in many systems to be critical links in the cascades from surface signals to the molecular alterations that result in functional plasticity. Chronic antidepressant treatments can regulate intracel

減至最低

Zeit-Strom-Verhalten bis zum Unterbrechen,e as endogenous modulators of complex behaviours, including anxiety-related behaviour and psychopathology, particularly due to their high number and diversity, the dynamics of release patterns in distinct brain areas and the multiple and variable modes of interneuronal communication they are involve

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