Titlebook: Antiarrhythmic Drugs; Mechanisms of Antiar Günter Breithardt,Martin Borggrefe,Gerhard Hindric Book 1995 Springer-Verlag Berlin Heidelberg 1

最低點(diǎn)

HERTZ

Experimental Models for the Assessment of Antiarrhythmic and Proarrhythmic Drug Action: Traditional may be defined as death which is nonviolent, unexpected, witnessed and instantaneous or occurs within a few minutes of an abrupt change in a previous clinical state [2]. Sudden cardiac death refers to such death with a cardiac cause, primarily ventricular fibrillation, and is a major health care pro

Dictation

Influence of the Autonomic Nervous System on the Action of Antiarrhythmic Drugsients, have eventually turned the interest toward arrhythmogenic interactions that often have not been foreseen or, at least, have not been thoroughly analyzed. As an example, when the Task Force of the Working Group on Arrhythmias of the European Society Cardiology called attention to the fact that

公社

incubus

SIT

音樂(lè)會(huì)

GENUS

Antiarrhythmic Drug Action: Modulation by Disease and Injury been disappointing. While some drugs can achieve high-grade suppression of extrasystoles, their effects on other components of vulnerability to sustained ventricular tachyarrhythmias may even result in a net increase in risk as demonstrated by the Cardiac Arrhythmia Suppression Trial (CAST) [9] and

劇毒

Truculent

Influence of the Autonomic Nervous System on the Action of Antiarrhythmic Drugs experimental observation that flecainide has the potential to actually favor the onset of life-threatening arrhythmias when acute myocardial ischemia is accompanied by sympathetic activation [2] (Fig. 1).