Titlebook: Antiarrhythmic Drugs; Mechanisms of Antiar Günter Breithardt,Martin Borggrefe,Gerhard Hindric Book 1995 Springer-Verlag Berlin Heidelberg 1

Osteoarthritis

Cocker

Use Dependence and Reverse Use Dependence of Antiarrhythmic Agents: Pro- and Antiarrhythmic Actionsod stops, while in tachycardia the heart contracts so frequently that it contracts before it has properly filled. As a result cardiac output is very low. Because of the seriousness of these two arrhythmias, I concentrate on the electropharmacology of agents that interfere with tachycardias and fibrillation.

加強(qiáng)防衛(wèi)

污穢

案發(fā)地點(diǎn)

Expediency

Ventilator

Sodium Channel Blockade as an Antiarrhythmic Mechanismly lead to safer, more effective treatment. This paper reviews the normal function of the sodium channel, the mechanism(s) of its blockade by drugs and the implication of the blocking mechanisms to the clinical use of these drugs.

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receptors

Experimental Models for the Assessment of Antiarrhythmic and Proarrhythmic Drug Action: Traditional Obviously, there is a need for an effective form of therapy capable of safely treating patients at risk of sudden cardiac death and therefore, a priori, a need for appropriate animal models for preclinical drug testing.

Indecisive

Book 1995t pharmacological treatment to a situation in which there is now marked uncertainty and general apprehension about the role of antiarrhythmic drugs. Until relatively recently the prevailing concept in antiarrhythmic therapy was that arrhythmias could be controlled by drugs which slowed conduction or