Titlebook: West Nile Virus; Methods and Protocol Tonya M. Colpitts Book 2016 Springer Science+Business Media New York 2016 West Nile Virus infection.i

indices

尖牙

A Brief Review of West Nile Virus Biology,NV is an arthropod-transmitted virus that mainly affects birds but humans become infected as incidental dead-end hosts which can cause outbreaks in na?ve populations. The main vectors of WNV are mosquitoes of the genus ., which preferentially feed on birds. As in many other arboviruses, the characte

表狀態(tài)

INTER

CONE

閃光你我

流浪

Lessons from the Murine Models of West Nile Virus Infection,r parts of the world over the past decade. Up to 50?% of WNV convalescent patients were reported to have long-term neurological sequelae or chronic kidney diseases. However, there are neither antiviral drugs nor vaccines available for humans. The underlying mechanism of the long-term sequelae is not

Foment

Exploration of West Nile Virus Infection in Mouse Models,ential cells can produce antiviral immune responses, the blood leukocytes are required to contain virus spread. However, infiltrating leukocytes may also contribute to immunopathology if they overreact. Thus analyses of WNV infectivity and leukocyte numbers in the CNS are critical for understanding

LUT

ALLAY

,In Vitro and In Vivo Blood–Brain Barrier Models to Study West Nile Virus Pathogenesis, molecular and cellular flux between the two. It plays a critical role in the maintenance of brain hemostasis. The BBB restricts the entry of pathogens into the brain, and thus its permeability is a critical factor that determines their central effects. Once the permeability of BBB is compromised, i