Titlebook: 25 Years of p53 Research; Pierre Hainaut,Klas G. Wiman Book 2005 Springer Science+Business Media B.V. 2005 DNA.P53, Wiman, Hainaut, tumor

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Heterogeneous Buffering for 3D NoCsversy and debate. Do these homologs behave like p53? Do they also act in tumor suppression? What were their origins – spin-offs of an ancestral p53 gene, or, in fact, predecessors of this famed ‘guardian of the genome’? . studies clearly reveal distinct physiological roles for p53, p63, and p73. But

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Introduction to 3D Technologieshe fact that the normally low levels of the p53 protein can be elevated in many cancers, it took some time to realise that p53 was in fact a tumour suppressor gene (Finlay et al., 1989; Levine et al., 1991). This concept was further consolidated by the discovery that the familial cancer predispositi

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Introduction to 3D TechnologiesMoll and Schramm, 1998), are just a few of the names that have been attributed to the p53 gene over recent years. However, the cameras (and funding) were certainly not present at the time of the discovery of p53 in 1979 (Crawford, 1983). It was only when the first alterations of the p53 gene in huma

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Zhenyu Xu,YeTong Wu,JunHong Zhongom/infotp.html; http;//www.sibiono.com) and p53 gene therapy gained regulatory approval in China in 2003 where it has been on sale since January 2004. Renewed optimism around the use of p53 gene therapy and increased understanding of p53 function suggest that many more potent and selective variants

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6GN for Future Wireless Networksion database at http://www.iarc.fr/p53). A majority of p53 mutations are missense mutations that give rise to the expression of mutant p53 proteins with one amino acid substitution. This pattern of mutation stands in sharp contrast to those of most other tumor suppressor genes, e.g. the Rb and p16 g