Titlebook: Viral Entry into Host Cells; Stefan P?hlmann,Graham Simmons Book 2013 The Editor(s) (if applicable) and The Author(s), under exclusive lic

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Viral Entry into Host Cells978-1-4614-7651-1Series ISSN 0065-2598 Series E-ISSN 2214-8019

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Picornavirus Entry,has become clear that entry is not a passive process, and that viruses initiate specific signals required for entry. And we have begun to recognize that for a given virus, there may be multiple routes of entry, depending on the particular target cell and the receptors available on that cell.

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Attachment Factors,required for their replication. The very large number of mammalian viruses identified to date is reflected in the fact that almost every human and animal cell type is a target for infection by one, or commonly more than one, species of virus. As viruses have adapted to target certain cell types for

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Picornavirus Entry,everal years progress has been made in understanding the structural changes in the virion important for uncoating and RNA release. In addition, for several viruses the endocytic mechanisms responsible for internalization have been identified, as have the cellular sites at which uncoating occurs. It

Condense

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Entry of Influenza Virus,, the virus first binds to cellularreceptors comprising either -(2,3 ) or -(2,6) linked sialic acid. Recent advances in our understanding of the influenza virus receptor and viral host species involved have shed light on the molecular mechanism of how influenza virus transmits across species and ada

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Paramyxovirus Entry,ns. Molecular and biochemical characterization over the past decade has revealed an extraordinary breadth of biological diversity among this family of viruses. Like all enveloped viruses, paramyxoviruses must fuse their membrane with that of a receptive host cell as a prerequisite for viral entry an