Titlebook: Ventricular Fibrillation and Sudden Coronary Death; M. E. Rajskina Book 1999 Springer Science+Business Media New York 1999 age.arrhythmia.

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M. E. Rajskina M.D., Ph.D.nzenes, monoterpenes and aldehydes were measured in August 1994 during the POPCORN campaign (POPCORN = .hoto-.xidant formation by .lant emitted .ompounds and .H .adicals in .orth-Eastern Germany). About 80 substances together contributed 90% of the atmospheric carbon in this range of molecular weigh

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M. E. Rajskina M.D., Ph.D.out 1% annually to 1.7 ppmV during the last decades (Khalil and Rasmussen, 1987). The resulting effect on global temperature is highly significant because the warming efficiency of methane is up to 30 times that of carbon dioxide (Dickinson and Cicerone, 1986). Data from polar ice cores indicate tha

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,The Ventricular Fibrillation and Heart’s Blood Supply,the higher the level of CAO, is the higher the probability that VF occurs. However, there is no answer to the question of why after a similar level of CAO some animals (and patients) develop VF but others do not.

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Ventricular Fibrillation and Changes in Metabolism of the Heart after Local Ischemia,tivity, automaticity and excitability. These changes are caused by changes in rest and action membrane potentials, which, in turn, are related to changes in K., Na. and Ca. ionic currents across the cell membrane.

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Mechanism of Ventricular Fibrillation Onset After Coronary Artery Occlusion, numerous metabolic hypotheses of the VF onset after CAO. The development of neuro-humoral hypotheses is based on new data about the role of the sympathetic and parasympathetic nervous system and their mediators (noradrenaline, adrenaline and acetylcholine) in the appearance of VF. A short review of these hypotheses is given below.

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Role of the Sympathoadrenal System in the Appearance of Ventricular Fibrillation after Coronary ArtIn the preceding chapter we showed that VF appears after CAO in cases where disturbances of metabolism in the ischemic zone occur at a higher rate and reach greater values than in cases without VF. A hypothesis was formed that this is due to more dramatic disturbances of the sympathoadrenal heart control in cases with VF.

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