Titlebook: Vascular Ion Channels in Physiology and Disease; Irena Levitan, PhD,Alex M. Dopico, MD, PhD Book 2016 Springer International Publishing Sw

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Mitochondrial Depolarization in Endothelial and Other Vascular Cells and Their Role in the Regulatioignaling pathways. Activation of the adenosine triphosphate (ATP)-dependent potassium channels on the inner mitochondrial membrane (mitoK. channels) leads to mitochondrial depolarization as well as activation of cell-specific signaling mechanisms in endothelium, vascular smooth muscle (VSM), and per

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Ca2+/Calmodulin-Gated Small- and Intermediate-Conductance KCa Channels in Cardiovascular Regulation:and thereby initiate endothelium-derived hyperpolarization (EDH)-type of arterial dilation. The physiological relevance of this system in-vivo is evidenced by the observation that genetically encoded loss of K.3.1 and K.2.3 caused channel-subtype specific cardiovascular phenotypes characterized by e

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Kv7 Potassium Channels as Therapeutic Targets in Cerebral Vasospasmty and mortality and for which current therapies are inadequate. Ion channels in the smooth muscle cells of the cerebral vasculature have been considered as important molecular targets for anti-vasospastic therapies, but, other than the calcium channel blocker nimodipine, no therapies directed towar

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Calcium Mobilization via Intracellular Ion Channels, Store Organization and Mitochondria in Smooth Mnternal Ca. stores containing IP.R and RyR may be arranged as multiple separate compartments with various IP.R and RyR arrangements, or there may be a single structure containing both receptors. The existence of multiple stores is proposed to explain several physiological responses which include the

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