Titlebook: Vascular Endothelium; Pharmacologic and Ge John D. Catravas,Allan D. Callow,Una S. Ryan Book 1998 Springer Science+Business Media New York

Waterproof

書目名稱Vascular Endothelium影響因子(影響力)




書目名稱Vascular Endothelium影響因子(影響力)學(xué)科排名




書目名稱Vascular Endothelium網(wǎng)絡(luò)公開度




書目名稱Vascular Endothelium網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Vascular Endothelium被引頻次




書目名稱Vascular Endothelium被引頻次學(xué)科排名




書目名稱Vascular Endothelium年度引用




書目名稱Vascular Endothelium年度引用學(xué)科排名




書目名稱Vascular Endothelium讀者反饋




書目名稱Vascular Endothelium讀者反饋學(xué)科排名

DIS

monopoly

Mechanisms of Endothelial Injury and Repairoducts such as the reactive oxygen species (ROS), proteases and the already mentioned cytokines. The effects of oxygen radicals will be discussed more specifically because they represent a well studied model of endothelial injury. They are also associated with other important factors implicated in microvascular damage.

Hemiparesis

Cytokine Regulation of Endothelial Cell Functionnd function of white blood cells require an intimate relationship with vascular EC. Cytokines are mediators of these complex bi-directional interactions between leukocytes and vascular elements (for review see Mantovani and Dejana, 1989, Mantovani, Bussolino, and Dejana, 1992).

Mitigate

Mechanisms of Endothelial Injury and Repairspiratory distress syndrome being a paradigm of such a complex situation where toxic and inflammatory elements interact. The role of cytokines has received prominent attention and their involvement in the expression of adhesion molecules has emphasized the importance of the intricate relationship be

秘方藥

MILL

仲裁者

Control of iNOS Expression in Rat Aortic Smooth Muscle Cellse NO synthase (iNOS or type II NOS) in a variety of cells, including vascular endothelial and smooth muscle cells.. This enzyme also uses L-arginine as substrate and is sensitive to inhibitory L-arginine analogues; furthermore, it does not appear to differ significantly from ecNOS on the basis of co

seduce

埋伏

Insertion/Deletion (I/D) Polymorphism in the Angiotensin Converting Enzyme (ACE) Gene the report by Cambien and his co-workers (Cambien et al., 1992) of association between the D allele and coronary heart disease (CHD), particularly in CHD patients without classical risk factors. In the following, this observation by Cambien’s group will be compared with observations in our group an