Titlebook: Understanding PTMs in Neurodegenerative Diseases; Victor Corasolla Carregari Book 2022 The Editor(s) (if applicable) and The Author(s), un

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,Post-translational Modifications in Parkinson’s Disease,ia nigra leading to severe motor complications. The etiology of the disease is unknown with its sporadic form accounting for 90% of cases. To date, over 20 genes have been identified as the cause of the inherited form of PD, many of them linked to the protein alpha-synuclein and mitochondrial functi

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Mitochondrial Dysregulation and the Influence in Neurodegenerative Diseases,urvival are closely related to proper mitochondrial function. Dysregulation of mitochondrial processes can lead to several detrimental effects in the cells and stablish the condition of mitochondrial dysfunction. This dysfunction is proposed to be greatly implicated in several neurodegenerative dise

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PTMs: A Missing Piece for Schizophrenia Studies,can undergo to change various physical and functional properties. PTMs hold great potential to better understand multifactorial diseases and disorders like schizophrenia. The field of PTMomics is still expanding and developing, though several modifications have already been implicated in the etiolog

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Post-translational Modifications in Brain Diseases: A Future for Biomarkers,l modification of proteins, however, is an important regulatory system of many biological processes, affecting a wide range of biochemical properties of proteins, including their binding, localization, activity, and stability. These modifications are not analyzed if not specifically searched for in

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Book 2022ional modifications (PTMs) can be applied to neurodegenerative diseases and relevant studies. The goal of the book is to increase awareness among researchers about how PTMs may be helpful in understanding mechanisms in various neurodegenerative diseases through proteomic studies. This book will serv

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Mitochondrial Dysregulation and the Influence in Neurodegenerative Diseases,cells and stablish the condition of mitochondrial dysfunction. This dysfunction is proposed to be greatly implicated in several neurodegenerative diseases, with evidence of compromised mitochondrial function and dynamics in Alzheimer’s disease, Parkinson’s disease, Huntington’s disease, and amyotrophic lateral sclerosis.

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