Titlebook: Understanding Glial Cells; Bernardo Castellano,Berta González,Manuel Nieto-Sa Book 1998 Springer Science+Business Media New York 1998 Alzh

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Role of Thyroid Hormone on the Oligodendrocyte Type 2-Astrocyte Lineageid animals, but the number of progenitors and astrocytes were unchanged. Next, to obtain insights into how the timely generation of these cells is regulated by thyroid hormone (T3) we have used an “in vitro” model of oligodendrocyte differentiation from pure cultures of O2A progenitor cells. Our res

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Characterization of Rat Neural Stem Cells from Embryonic Striatum and Mesencephalon During in Vitro he levels of cyclic AMP, during in vitro neural stem cells differentiation. We have also studied the endogenous content of amino acids which are well known as trophic factors. Significant levels of glutamate, taurine, and glycine are found in neural stem cells at any time studied (3, 10, and 17 days

DRAFT

Reservation

Cytokines in Astroglial Cells: Functions and Mechanisms of Actione interleukin-1 (IL-1α and β), interleukin-6 (IL-6), tumor necrosis factor-α (TNF-α), and interferon γ (IFN-γ). We review here the production of immunoinflammatory mediators like nitric oxide and prostaglandins as well as cytokines such as IL-6 and TNF-α by cultured astrocytes upon IL-1α or β stimul

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Ethanol Exposure During Brain Development Alters Astrogliogenesis and Astrocyte Functions in primary cultures), that prenatal alcohol exposure induces important alterations in the astrogliogenesis. In fact, several groups including ours have demonstrated that alcohol induces decreases in either DNA synthesis or cell proliferation, decreases in protein synthesis and content, decreases in

Antimicrobial

Neuron-Glia Ensembles and Mammalian CNS Lesion Repair also permit to correct the consequences of major CNS injury. True regeneration, i.e. the restitution of the CNS to its pre-injury state, is clearly impossible in the absence of neuronal division. However, we think that functional repair can be achieved. The present chapter describes, within the abo

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Effects of Contralateral Lesions and Aging on the Neuronal and Glial Population of the Cerebral Cortaddition, pericytes and perivascular microglial cells accumulated a great number of dense and foamy inclusions during aging. Finally, both neurodegenerative processes, lesion and aging, seems to produce glial hyperplasia and hypertrophia through a different mechanism which also differentially affect