Titlebook: Underactive Bladder; Christopher R. Chapple,Alan J. Wein,Nadir I. Osman Book 2017 Springer International Publishing Switzerland 2017 Allie

MELON

想象

Epidemiology of Underactive Bladder,he symptoms and signs of DU have some crossover with those associated with Bladder outlet obstruction (BOO). This poses a considerable challenge to the acquisition of useful epidemiological data as it is impractical to perform urodynamic studies on large groups of people who may be entirely asymptom

SPER

有組織

Pharmacological Treatment of Underactive Bladder,chol and carbachol to stimulate detrusor muscarinic receptors, choline esterase inhibitors, like distigmine to reduce the degradation of acetylcholine, and α-adrenoceptor antagonists to block α-adrenoceptors in the outflow region. α-Adrenoceptor antagonists may have a beneficial effect on UAB/DUA in

女歌星

Conquest

Neurostimulation and Neuromodulation for the Treatment for the Underactive Bladder,r different neurogenic situations such as the anterior sacral root stimulation after selective posterior sacral rhizotomy (the Brindley technique) in patients with a complete spinal cord injury and preserved contractility of the detrusor. Sacral neuromodulation is clinically available and FDA approv

招惹

Reduction Cystoplasty,unctional outcomes seems counterintuitive, the premise can be explained in relation to Laplace’s law. This law states that P = k*T/R, where P is the cavitary pressure, k is a constant, T is the wall tension, and R is the radius of the chamber. Assuming wall tension remains constant, intraluminal voi

Anticonvulsants

Jocose

Tissue Engineering and Cell Therapy for Underactive Bladder: Current and Future Approaches,eering of whole bladders has been investigated for over a decade and resulted in a small clinical study with some success, however such bladders can only fulfill a storage function due to the difficulty of recreating the complex neural innervation to enable volitional emptying. Stem cell injection t

Mawkish

Reflections and the Way Forward,he understanding of the underlying pathophysiological basis of these symptoms. For the most part there has been an overwhelming focus on detrusor overactivity (DO) and bladder outlet obstruction (BOO) as the causes storage and voiding LUTS respectively. This focus has been beneficial in that it has