Titlebook: Topics in Pathophysiology of Hypertension; Herman Villarreal (Head),Mohinder P. Sambhi (Profe Book 1984 Martinus Nijhoff Publishers, Bosto

一回合

Biomembrane Abnormalities in Spontaneous Hypertensionews is that hypertension and stroke are definitely determined by genetic factors [4]. On the other hand, the good news is that hypertension and stroke can be prevented by modifying environmental factors or by preventive treatments, even if genetic predisposition is strong [3, 5, 6].

pulmonary

Erythrocyte Na+ Handling in Rat Genetic Hypertension. Transmembrane pathways in the red cells of genetically hypertensive rats are also altered [2]. This paper summarizes our previous findings on Na. transport systems in SHR and SbH and reports some new data on the effect of chronic and acute Na. loads on cellular Na. handling.

Monotonous

CA2+ Binding by the Erythrocyte Membrane and its Structure in Primary and Secondary Hypertensionspontaneous hypertension of rats (SHR, Kyoto-Wistar). In particular, it was demonstrated in both forms of primary hypertension that permeability of the erythrocyte membrane for monovalent cations is increased [1–3], and the calcium-binding ability of the inner part of the membrane is decreased [4, 5

FLEET

The Na+/K+ Co-Transport System in Erythrocytes from Essential Hypertensive Patientsnic movements are allowed by genetically coded transport systems. Regarding Na. and K. transport, most of the carrier-mediated fluxes are catalyzed by the Na./K. pump [2]. This system catalyzes the exchange of intracellular Na. by extracellular K. coupled to the hydrolysis of ATP, thus generating el

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