Titlebook: Sunlight, Vitamin D and Skin Cancer; J?rg Reichrath Book 2008 The Editor(s) (if applicable) and The Author(s), under exclusive license to

細(xì)胞膜

儲(chǔ)備

Molecular Biology of Basal and Squamous Cell Carcinomas,arcinomas. Significant progress has also been made in our understanding of squamous cell carcinomas of the skin. In this chapter, we will focus on major recent developments in our understanding of basal cell carcinomas and squamous cell carcinomas at the molecular levels and their clinical implications.

Intellectual

Sunscreens,iation (UVBR) is about 1000 times more erythemogenic than short-wave UVAR. Under normal conditions, middle and long-wave UVAR do not induce sunburn. UVAR has therefore been further broken down into two bands UVA1 (340–400 nm) and UVA2 (320–340 nm) because of the increased erythemogenic activity of UVA2 compared to UVA1.

維持

Substance-Abuse

Melanoma and Nonmelanoma Skin Cancers and the Immune System,up on the part of the tumor cells? What circumstances lead to spontaneous regression vs. metastatic malignancy? Furthermore, if tumor cells originate from cells that were previously seen as “self” what is the significance of autoimmunity and regulation of these “surveillance” mechanisms?

叢林

outskirts

認(rèn)識(shí)

決定性

Vitamin D Status and Cancer Incidence and Mortality,potential role of sun exposure in reducing risk of several types of internal cancers. The hypothesis that sunlight may be beneficial against several types of cancer extends back almost seven decades. Initially, Peller and Stephenson observed higher rates of skin cancer, but lower rates of other mali

GEST

Sun Exposure and Cancer Survival in Norway: Changes in the Risk of Death with Season of Diagnosis artant source of vitamin D. Seasonal variations and latitudinal gradients of calcidiol (the marker of vitamin D status) have been reported. We wanted to investigate if season and latitude play any role for survival from seven different cancer types in Norway. Seasonal and geographical variations of v