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Titlebook: Stroke Revisited: Dyslipidemia in Stroke; Seung-Hoon Lee,Min Kyoung Kang Book 2021 The Editor(s) (if applicable) and The Author(s), under

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發(fā)表于 2025-3-21 17:20:16 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Stroke Revisited: Dyslipidemia in Stroke
編輯Seung-Hoon Lee,Min Kyoung Kang
視頻videohttp://file.papertrans.cn/880/879759/879759.mp4
概述Presents state of the art knowledge.Covers both basic and clinical aspects in detail.Evaluates currently available treatment modalities.Discusses a range of specific considerations.Provides clear mana
叢書(shū)名稱(chēng)Stroke Revisited
圖書(shū)封面Titlebook: Stroke Revisited: Dyslipidemia in Stroke;  Seung-Hoon Lee,Min Kyoung Kang Book 2021 The Editor(s) (if applicable) and The Author(s), under
描述This book presents state of the art knowledge on dyslipidemia in stroke, covering both basic and clinical aspects in detail. The focus is in particular on two major themes: the clinical significance of dyslipidemia in stroke and the treatment of dyslipidemia in stroke patients. Readers will find up-to-date information on lipid metabolism, biomarkers, and advances in treatment options, including novel biologic drugs. Specific management considerations and pitfalls are also discussed. The individual components of pathophysiology, treatment, and key issues are addressed with the aid of complementary illustrations that facilitate understanding of practical aspects and enable the reader to retrieve fundamental information quickly. This book is timely in bringing together within one volume the most important current knowledge on dyslipidemia in stroke. It will be invaluable for stroke physicians, pharmacists, and students seeking to acquire up-to-date knowledge..
出版日期Book 2021
關(guān)鍵詞dyslipidemia; stroke; atherosclerosis; cardiovascular disease; biomarker
版次1
doihttps://doi.org/10.1007/978-981-16-3923-4
isbn_softcover978-981-16-3925-8
isbn_ebook978-981-16-3923-4Series ISSN 2522-5588 Series E-ISSN 2522-5596
issn_series 2522-5588
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature Singapor
The information of publication is updating

書(shū)目名稱(chēng)Stroke Revisited: Dyslipidemia in Stroke影響因子(影響力)




書(shū)目名稱(chēng)Stroke Revisited: Dyslipidemia in Stroke影響因子(影響力)學(xué)科排名




書(shū)目名稱(chēng)Stroke Revisited: Dyslipidemia in Stroke網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱(chēng)Stroke Revisited: Dyslipidemia in Stroke網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




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發(fā)表于 2025-3-21 23:13:42 | 只看該作者
Book 2021r on two major themes: the clinical significance of dyslipidemia in stroke and the treatment of dyslipidemia in stroke patients. Readers will find up-to-date information on lipid metabolism, biomarkers, and advances in treatment options, including novel biologic drugs. Specific management considerat
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地板
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發(fā)表于 2025-3-22 12:32:00 | 只看該作者
2522-5588 usses a range of specific considerations.Provides clear manaThis book presents state of the art knowledge on dyslipidemia in stroke, covering both basic and clinical aspects in detail. The focus is in particular on two major themes: the clinical significance of dyslipidemia in stroke and the treatme
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發(fā)表于 2025-3-22 16:58:05 | 只看該作者
Role of Dyslipidemia in Atherosclerosisue to the development of intimal plaques. The formation of plaques involves the deposition of small cholesterol crystals in the intima and its underlying smooth muscle. The growth of plaques starts with the proliferation of fibrous tissues and the surrounding smooth muscle producing a bulge inside t
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發(fā)表于 2025-3-22 20:39:59 | 只看該作者
Effects of Dyslipidemia on the Cerebral Vesselsone of the modifiable risk factors for stroke and increases the risk of stroke by 2.19 times [.]. Cholesterol can be largely divided into total cholesterol, triglyceride (TG), low-density lipoprotein (LDL), and high-density lipoprotein (HDL). When the endothelial cell of blood vessels is damaged by
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Clinical Biomarkers of Dyslipidemiactors like total cholesterol LDL cholesterol and HDL cholesterol. However, apolipoproteins like Apo B100 sometimes can be better predictor for assessing the risk of the cardiovascular disease and stroke. The stroke pathomechanism is heterogeneous and multifactorial. Oxidative stress involves the inj
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