Titlebook: Spontaneous Intracerebral Haematomas; Advances in Diagnosi Hans Werner Pia (Direktor),Charles Langmaid,Jan Zi Conference proceedings 1980 S

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Conclusions,f bleeding with the broad screening of all possible factors, in particular, investigation of the dyscrasias and diagnosis of hypertension. This requires the help of a team, including haematologists and cardiovascular physicians.

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Special Lecture,roke” may be classified as an “abrupt, sudden, or progressive neurological defect syndrome (of consciousness motor, sensory, neuropsychological and/or other systems) on a vascular basis”. Apparently this goes further than the semantic content of the term “stroke”, which means literally that some ind

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Morphology and Aetiology,tors do not play an essential part (D. Russell 1954) although haemorrhages from ruptured aneurysms and arteriovenous malformations have also been excluded by some authors (Johansson and Melin 1960; Walter and Schütte 1965; Vanderark and Kahn 1968; Benes et al. 1972), as have intracerebral haematomas

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Clinical Pathology,e brain’s autoregulatory capacity and by this effect alone reduce cerebral blood flow. As local perfusion pressure may vary and at least is unpredictable, regional cerebral blood flow is probably regulated by mechanisms which are even more difficult to define than the normal functional metabolic lin

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Aetiological Aspects of Location,frequent sites of intracerebral bleeds are in the basal ganglia and cerebral hemispheres and of these, 60%–80% rupture into the ventricles (Mutlu et al. 1963; Yates 1976; Hurtig and Reivich 1977; Jellinger 1977). Haemorrhages into the basal ganglia account for 40%–66% of all primary intracerebral bl

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Diagnosis,is reason, pre-operative investigations, particularly contrast studies, have a very important part to play. Computer tomography has enriched the process of diagnosis in many ways (Terao et al. 1979). However, even today in individual cases the assessment and differential diagnosis can still be quite

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