Titlebook: Spin Dynamics in Two-Dimensional Quantum Materials; A Theoretical Study Marc Vila Tusell Book 2022 The Editor(s) (if applicable) and The Au

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Book 2022odels and state-of-the-art quantum transport methodologies. The main outcomes of this work are: (i) the analysis of the crossover from diffusive to ballistic spin transport regimes in ultraclean graphene nonlocal devices, and (ii) investigation of spin transport and spin dynamics phenomena (such as

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,Quantum Transport Methodologies for?Spin Transport,cal equations, they offer unique advantages. One can calculate properties without performing an experiment and thus it is possible to make predictions about uncharacterized materials, undiscovered physical effects or obtain microscopic insights into physical phenomena that experiments cannot unveil.

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2190-5053 pins go ballistic.Describes spin transport experiments on a .This thesis focuses on the exploration of nontrivial spin dynamics in graphene-based devices and topological materials, using realistic theoretical models and state-of-the-art quantum transport methodologies. The main outcomes of this work

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2190-5053 the thesis provides an in-depth theoretical interpretation of puzzling huge measured efficiencies of the spin Hall effect in MoTe.2., as well as a prediction of a novel canted quantum spin Hall effect in WTe.2. with spins pointing in the yz plane..978-3-030-86116-2978-3-030-86114-8Series ISSN 2190-5053 Series E-ISSN 2190-5061

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ty, formulation issues, pharmacokinetic and pharmacodynamic profiles. Nevertheless, we believe that this collection of 11 chapters by established leaders in the area of apoptosis will be of great interest to no978-94-007-9493-1978-94-007-4917-7

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e activation of growth factor receptors and their associated kinases or through formation of tyrosine kinase oncogenes. Overexpression and activation of receptor tyrosine kinases (RTKs) has been found in non small cell lung cancer (NSCLC) cells, including epidermal growth factor receptor (EGFR), HER

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Marc Vila Tusell DNA damage. In a similar pathway, damage-dependent phosphorylation of the checkpoint proteins Chk1 and Chk2 ultimately results in the radiation-inducible arrest of cells in G. through their downstream effects on Cdc25C. Likewise, ATM and ATR regulate the repair of DNA damage through phosphorylation

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