Titlebook: Signaling Through the Cell Matrix; Alvaro Macieira-Coelho Book 2000 Springer-Verlag Berlin Heidelberg 2000 Activation.cancer.cell.cell bio

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Book 2000 cell phenotype. For a long time the extracellular matrix was believed to have only a supporting role for cell attachement. However, it became apparent that the matrix participates actively in cell metabolism. The experiments that led to this conclusion are described in this volume. Progressively, m

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Collagen Type I: A Substrate and a Signal for Invasion,tly migration through the ECM defects (Bruyneel and Mareel 1981; Liotta et al. 1983). Noncancer cells invade the ECM in physiological and pathological situations, for example, during embryogenesis, during wound healing, and when cells are recruited into inflammatory regions.

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The Transmission of Contractility Through Cell Adhesion,heir neighbors. These mechanical interactions between groups of cells and the ECM, which joins these cells together, are important in tissue structure maintenance, physiological organ function, morphogenesis, and wound healing.

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Topological Constraints Carry Signaling from the Cell Matrix to the Genome,er cell (Macieira-Coelho et al. 1982). The small genomic modifications created by cell division are cumulative and become apparent morphologically, structurally, and metabolically (Macieira-Coelho 1988a).

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Role of Focal Adhesion Kinase in Signaling by the Extracellular Matrix,s, wound healing, and malignant transformation. The major cell surface receptors for ECM are the integrin family cell adhesion molecules which are composed of noncovalently associated α and ? subunits (Hynes 1992). Both α and ? subunits are transmembrane proteins containing short cytoplasmic domains

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