Titlebook: Sepsis and Organ Dysfunction; ...from Chaos to Rat Organ Failure Academy,A. E. Baue (Emeritus Profess Conference proceedings 2002 Springer-

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Alveolar Epithelium in Host Defence: Cytokine Productioneobronchitis, bronchiolitis and pneumonia, are significant clinical problems. The increasing number of multidrug-resistant microbes has made the treatment of these infections much more difficult [1]. To further improve therapies for respiratory infection, we need to learn more about the host defence

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Phagocytosis and Lung Injuryof the pathogen. However, while phagocytosis is a crucial element of host defence, aspects of the phagocytic response can also be associated with host tissue damage. I will review our current understanding of the basic mechanisms of phagocytosis, and potential sources of deleterious effects on the h

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Epithelial Injury in Sepsis and ARDS: Role of Leukocyte-Derived Proteases not the primary initiating factor directly induces acute inflammation (sepsis, disseminated intravascular coagulation, or pancreatitis) or whether there is direct injury to the lungs (hyperoxia or chemical injury). In the latter circumstances, although the initial injury may involve direct cellular

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The Role of Interleukin-10 During Systemic Inflammation and Bacterial Infectionems, including the cytokine network, is considered to play an important role in the pathogenesis of sepsis. Cytokines are a family of small proteins which function in a complex network in which they can influence each other’s production and activity. Pro-inflammatory cytokines, of which tumour necro

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Cardiovascular Surgery. Modulation of the Inflammatory Reactionac surgery. Incomplete myocardial protection is the cause of ischaemic complications and/or low cardiac output. CPB is responsible for systemic inflammatory response syndrome (SIRS), which can lead to multi-organ failure (MOF) [1].

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