Titlebook: Respiratory Control; A Modeling Perspecti George D. Swanson,Fred S. Grodins,Richard L. Hughs Book 1989 Plenum Press, New York 1989 Multivar

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Oxygen Kinetics in the Elderlylated losses appear greatest in pursuits requiring the integrated activity of a number of organ systems. The response to exercise exemplifies such an integrated response of control mechanisms, particularly during the dynamic, non-steady state phases of the adjustment to changing energy demands. The

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Neurogenic and Cardiodynamic Drives in the Early Phase of Exercise Hyperpnea in Manrs. The first purpose of the present study was to evaluate the quantitative role of both drives by comparing the ventilatory and circulatory responses to voluntary, electrically induced and passive exercise in healthy males and spinal cord transected patients with an equivalent work load.

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The Validity of the Cardiodynamic Hypothesis for Exercise Hyperpnea in Man Miyamoto et al.. have determined the kinetics of Q by adopting an ensemble-averaging technique to impedance cardiography, evidently showing that the change in Q precedes that in VE during the unsteady- state, of step, impulse and sinusoidal exercise.. However, the mechanism which links CO. flow to hyperpnea remains uncertain.

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Evidence for Possible ‘Cardiogenic’ Respiratory Drives in Exercising Manrved effects on breathing rather than the experimental stimulus itself. Thirdly, changes in the circulation need to be small, because reflex effects of arterial pressure on breathing through the carotid sinus have been established in man ..

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Control of Ventilation During Heavy Exercise in Man bronchial asthmatics,. and a substantial and persistent ventilatory depression with hyperoxic suppression of the carotid body during heavy exercise. have been the usual reasons cited for this position. This position has been challenged by many recent studies.

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On Smoothing Gas Exchange Data and Estimation of the Ventilatory Thresholdto the physiological variability and measurement error found in the alveolar gas exchange estimates. Thus, any technique which can reduce variability in the alveolar gas exchange estimates, should improve the precision of the VT estimates (7). Recent work in this area has attempted to reduce the phy

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A General-Purpose Model for Investigating Dynamic Cardiopulmonary Responses During Exerciseting it to espouse solely any particular control-system hypothesis, and (b) it allow progressive incorporation of particular observed behavior, thus developing in complexity (and completeness) in a “building-block” fashion. We found that surprising consequences often attended the simplest of stimulu