Titlebook: Researching Audio Description; New Approaches Anna Matamala,Pilar Orero Book 2016 The Editor(s) (if applicable) and The Author(s) 2016 Audi

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https://doi.org/10.1057/978-1-137-56917-2Audiovisual translation; media accessibility; audio description; reception studies; applied linguistics;

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2947-5740 rmation contained in the contributions identifies trends in current research-funded studies which will be valuable as a pointer towards future proposals. The bo978-1-137-56917-2Series ISSN 2947-5740 Series E-ISSN 2947-5759

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Gian Maria Grecoular studies have defined the role of these MHC products in controlling cellular interactions in the immune response both in T cell killing (class I products or classical histocompatibility antigens) and in lymphocyte—lymphocyte and lymphocyte—accessory cell interaction (class II products or la anti

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up looking for polymorphism and genetic markers. It just means that the quality of the genetic markers you get is much better. The recombination frequencies between the β-globin, for example, and the Hpa I site should be very small. No one has yet observed a recombinant despite about 1000 tests. Th

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Jana Holsanovaisjunction occurred in the female, then all of the X’s would come from the male; in this case you would have no Duchenne since it is an X- linked lethal. There is also a possibility of extreme lyonization which could also cause Duchenne in females if, by chance, all or the vast majority of their nor

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Iwona Mazur,Agnieszka Chmielable 2). More recent linkage studies with classic genetic markers such as color blindness and G6PD deficiency have provided consistent evidence of the presence of a major single gene located on the distal end of the long arm of the X-chromosome in bipolar manic-depression (Mendlewicz 1986,1988), des

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José Dávila-Montes,Pilar Oreroe for elevated CT and may contribute as much as one-fifth of the attributable risk for systolic hypertension. Genetic animal models for hypertension may not reflect the genes operative in human hypertension..Hypertension is a common trait with familial aggregation (see Ward 1990; Burke and Motulsky

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Nazaret Fresno,Judit Castellà,Olga Soler-Vilagelius strong and due mostly to genetic factors rather than shared family environment. As expected, lipid abnormalities were up to five times more common among coronary siblings than in the general population, but hypertensive siblings also showed up to four times more frequent lipid abnormalities than t