Titlebook: Regulatory Mechanisms of Synaptic Transmission; Ricardo Tapia,Carl W. Cotman Book 1981 Plenum Press, New York 1981 brain.catecholamines.mu

Aspirin

有權(quán)

Ballerina

Calcium, Calmodulin, and Synaptic Function: Modulation of Neurotransmitter Release, Nerve Terminal smission and the action of specific neuropharmacologic agents, and possibly provide new insights into human disease processes involving synaptic modulation. Although calcium’s role in neurotransmitter release from the presynaptic nerve terminal has been of great interest, little is known about the m

Urgency

Urgency

Effect of Nerve Growth Factor on Microtubules: Evidence for a Protective Action against Depolymerizn about its molecular mechanisms and physiological action. This is frequently the case in the field of hormones and growth factors, whose phenomenological aspects (spectrum of actions, number and type of target cells, etc.) become known even decades before the understanding of their mechanism of act

Hyperplasia

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Presynaptic Control of Information Transmission in the Vertebrate Spinal Cord,th the ., in its broadest sense. The problem is to define what do we mean by regulation, and this is not an easy task. In this context we may distinguish two types of mechanisms controlling synaptic transmission. One, involving actions tending to maintain a . operating level in the system, for examp

ANTH

Synaptic Changes Induced by Optic Chiasm Low Intensity Repetitive Electrical Stimulation (The Kindlminates in the appearance of electrographic and behavioral convulsions (9). Kindling has been considered as a model for learning, long term memory, and plasticity (16). Many brain regions are unresponsive to the kindling effect, specifically the majority of the neocortex, the thalamus and the brain

Ergots

fatty-acids