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Titlebook: Regression of Atherosclerotic Lesions; Experimental Studies M. Rene Malinow,Victor H. Blaton Book 1984 Springer Science+Business Media New

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書目名稱Regression of Atherosclerotic Lesions
副標(biāo)題Experimental Studies
編輯M. Rene Malinow,Victor H. Blaton
視頻videohttp://file.papertrans.cn/826/825520/825520.mp4
圖書封面Titlebook: Regression of Atherosclerotic Lesions; Experimental Studies M. Rene Malinow,Victor H. Blaton Book 1984 Springer Science+Business Media New
描述The Advanced Study Institute on Regression of Atherosclerotic Lesions, sponsored by the North Atlantic Treaty Organization, was held in Maratea, Italy, September 11-13, 1983. The institute was designed to foster discussion of experimental data on the regression of atherosclerosis in animals and evidence of a similar phenomenon in humans. The participating scientists showed with anatomical and biochemical data, that atherosclerosis regresses in cholesterol fed animals after withdrawal of atherogenic stimuli. The existence of regression in humans was inferred from sequential angiographic visualization of arteries, but anatomical and biochemical characteristics of the process have not been defined. Two mechanisms are probably involved in such an angiographic regression. The first, which may occur when hyperlipidemia is partly or fully corrected or during undernutrition or other metabolic abnormalities, may be related to atherosclerosis changes noted in animals undergoing regression. The second type seems independent of changes in plasma lipid levels and may involve resolution of thrombi or other underlying arterial wall factors. The relative frequencies of the two mechanisms are unkno
出版日期Book 1984
關(guān)鍵詞Lipoprotein; artery; atherosclerosis; drugs; evolution; experiment; high-density lipoprotein (HDL); nutriti
版次1
doihttps://doi.org/10.1007/978-1-4757-1773-0
isbn_softcover978-1-4757-1775-4
isbn_ebook978-1-4757-1773-0
copyrightSpringer Science+Business Media New York 1984
The information of publication is updating

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Progression and Regression of Nonhuman Primate Coronary Artery Atherosclerosis: Considerations of Exnly 3% of the atherosclerosis studies supported by their Institute dealt with regression of atherosclerosis. Two years later, Armstrong and his co-workers described the regression that occurred in diet-induced coronary artery atherosclerosis of rhesus monkeys when the diet was changed from one high
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The Comparative Pathology of Nonhuman Primate Atherosclerosisof the disease in late middle and old age. The atherosclerotic lesions of the ideal model would range from trivial abnormalities, such as fatty streaks, found only at necropsy, to raised plaques with complications such as ulceration, stenosis, hemorrhage and superimposed thrombosis. The number of an
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Plant Glycosides: Effects on Atherosclerosis Regression in ,ates stemmed from the observations of Armstrong et al. in rhesus macaques (.).. They demonstrated that in advanced coronary atherosclerotic lesions induced by a high-cholesterol diet there is a remarkable decrease in lumen stenosis when the animals are shifted to a low-cholesterol diet. Their observ
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Use of Cholesteryl Ester Analogs to Study Transport of Lipoproteins in Intact Animalslem of quantitation of the lipids present at the various stages of regression. During the past few years we have synthesized labeled analogs of cholesteryl ester (1) and triacylglycerol (2,3) which owing to the replacement of the ester bond by an ether bond are not hydrolyzed by the esterases presen
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