Titlebook: Reelin Glycoprotein; Structure, Biology a S. Hossein Fatemi (Professor of Psychiatry, Adjunc Book 2008 Springer-Verlag New York 2008 Alzhei

BARK

S. Hossein Fatemi (Professor of Psychiatry, AdjuncProvides an unprecedented analysis of this emerging and novel protein by examining evidence from genetic, neuroanatomic, biochemical, and behavioral studies

dearth

alliance

幾何學(xué)家

PALMY

Book 2008roduct could cause abnormal cortical development and reelin signaling impairment in brain. Reelin abnormalities in several neurospychiatric disorders, such as autism, schizophrenia, mood disorders and lissencephaly, may provide mechanistic explanations for etiologies of these disorders. There is als

證明無(wú)罪

The , Gene and Its Functions in Brain Development, functions have been elucidated by the analysis of mutant mice and biochemical interactions, but much remains to be discovered. In this chapter we will summarize the current state of our knowledge of . and its function in brain development.

內(nèi)向者

Crystal Structure of Reelin Repeats,mology between subrepeat A and subrepeat B was first noted during the cloning of the reelin gene (D’Arcangelo ., 1995), these repeats have failed to show any sequence homology to known protein domains.

Madrigal

Reelin/Dab1 Signaling in the Developing Cerebral Cortex,g mechanisms of Reelin signaling. What has emerged from these studies is a surprisingly complex picture of Dab1 at the genomic, mRNA, protein, and functional levels. This chapter will summarize some of the key features of Dab1, and its role as a transducer of the Reelin signal in the developing cerebral cortex.

Ingrained

Reelin, Liver, and Lymphatics,in may have on peripheral organs, however, remains very scarce. Reelin mRNA and protein have been detected during development and adulthood in several peripheral organs. In the present chapter, we will focus on the presence of reelin in liver and lymphatics and discuss some hypotheses about the functional significance of this presence.

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