Titlebook: Redox-Active Therapeutics; Ines Batini?-Haberle,Júlio S. Rebou?as,Ivan Spasoj Book 2016 Springer International Publishing Switzerland 2016

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Superoxide Dismutase Family of Enzymes in Brain Neurogenesis and Radioprotection,ineage, while EC-SOD deficient mice exhibited a significant reduction in the production of newborn neurons. Similarly, ionizing radiation, even at low doses, led to persistent perturbation of the tissue redox environment and suppression of hippocampal neurogenesis. The defects in hippocampal neuroge

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Mn Porphyrin-Based Redox-Active Therapeutics, ischemia/reperfusion injuries of liver and kidney. Based on their ability to mimic kinetics and thermodynamics of the catalysis of superoxide (O..) dismutation, the cationic . Mn(III) .-substituted pyridylporphyrins (MnPs) were identified as the most powerful mimics. Over years the general knowledg

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Nitrones as Potent Anticancer Therapeutics,mor growth in a transgenic mouse model for colorectal polyps. Others have also shown that PBN and the PBN-analogue, CQPN (C-(2-chloroquinoline-3-yl)-.-phenyl nitrone) are effective in inhibiting tumor growth in a rat mammary tumor model, and breast carcinoma cell growth, respectively. The mechanism-

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Salen Manganese Complexes Mitigate Radiation Injury in Normal Tissues Through Modulation of Tissue tors of oxidative stress are increased in lung and skin radiation injury models, and suppressed by salen Mn complexes. Furthermore, histological, gene expression, and other assessments suggest that salen Mn complex treatment promotes a more normalized tissue environment in irradiated animals, includ

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Molecular Basis for Anticancer and Antiparasite Activities of Copper-Based Drugs,cell, and efficiently induce apoptosis in different tumor cells, having DNA and mitochondria as main targets. Additionally, this kind of metal-based compounds can inhibit specific proteins, implicated in topological changes of DNA, such as topoisomerases, or in the control of cell cycle, such as kin

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