Titlebook: Recent Developments in Toxicology: Trends, Methods and Problems; Proceedings of the E Philip L. Chambers,Claire M. Chambers,Siegfried Go Co

指耕作

FLIT

Neurochemical Characterization of Hexachlorophene Induced Brain Oedemasly within some weeks after termination of drug treatment. The aim of the present study was to provide an additional neurochemical characterization of hexachlorophene oedema at the paralytic climax of its occurrance. The parameters used were

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裂隙

The Effects of n-Hexane and its Metabolites on Erythrocyte and Synaptosome Membrane Acetylcholinestetransport through the membrane (Korpela and T?hti 1986). In the present study, the effect of n-hexane and its metabolites 2,5-hexanedione, 2-hexanol and 2-hexanone on membrane acetylcholinesterase (AChE) were studied.

朦朧

Assessment of Toxicopathological Effects in Ageing Laboratory Rodentsckground of natural ageing changes. The following issues are of particular concern: normal “age-related” data, consistency and relevance of pathology data, detection of subtle changes, age-related susceptibility to toxicants.

GREG

Tumor Registry Data Base: Systematized Nomenclature of Preneoplastic and Neoplastic Lesions in Rat Gnd from several chemical and pharmaceutical companies in West Germany and Switzerland are collaborating to set up a data base for preneoplastic and neoplastic lesions. For this purpose the nomenclature applied to various lesions was systematized. This paper illustrates some of the criteria employed

white-matter

Spangle

cornucopia

The Glutathione System in Rat Liver Chronically Injured by Thioacetamidezschmar and Klinger 1990). GSH is transported into the circulation through the sinusoidal membrane by a carrier transport system (Kaplowitz et al 1985). This fact is important for supplying extrahepatic tissues with GSH or cysteine (Bannai and Tataishi 1986). Recently, Burgunder and Lauterburg (1987

vertebrate

Hitherto Unknown Additive Growth Effects of Fluorene and 2-Acetylaminofluorene on Bile Duct Epitheliity of a compound, its potency to cause cell proliferation is considered to be most reliable. However, most genotoxic carcinogens inhibit division of their target cells. This occurs even in the early phase after non-necrogenic doses. Contrarily, non-genotoxic carcinogens of different organotropism e