Titlebook: Recent Advances in Epilepsy Research; Devin K. Binder,Helen E. Scharfman Book 2004 The Editor(s) (if applicable) and The Author(s), under

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書目名稱Recent Advances in Epilepsy Research影響因子(影響力)




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書目名稱Recent Advances in Epilepsy Research被引頻次學(xué)科排名




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0065-2598 gic and phenotypic characterization. .Recent Advances in. .Epilepsy Research. sets forth a series of chapter reviews by researchers involved in these advances. This volume is a composite profile of some exciting recent investigations in select areas of enquiry. .Key features: neurogenetics of seizur

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Vascular Endothelial Growth Factor (VEGF) in Seizures:,eability, and inflammation. In brain, receptors for VEGF have been localized to vascular endothelium, neurons, and glia. VEGF is upregulated after hypoxic injury to the brain, which can occur during cerebral ischemia or high-altitude edema, and has been implicated in the blood-brain barrier breakdow

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Role of the GABA Transporter in Epilepsy,t largely from blocking this reuptake. However, there is another side to the GABA transporter, contributing to GABA release by reversing in response to depolarization. We have recently shown that this form of GABA release is induced by even small increases in extracellular [K.], and has a powerful i

MERIT

GABA and Its Receptors in Epilepsy,s that are ligand-operated on channels and the G-protein-coupled metabotropic GABA. receptors. Impairment of GABAergic transmission by genetic mutations or application of GABA receptor antagonists induces epileptic seizures, whereas drugs augmenting GABAergic transmission are used for antiepileptic

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Role of the Depolarizing GABA Response in Epilepsy,ion of the nervous system. Thus 2 important criteria for a candidate convulsant mechanism are that the mechanism is compatible with normal neural functioning, and that the mechanism can produce rapid deviations from normal function. For example, the blockade of GABA-mediated inhibition is a robust a