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Titlebook: Rb and Tumorigenesis; Maurizio Fanciulli Book 2006 Springer-Verlag US 2006 DNA.cell.cell cycle.enzyme.enzymes.gene.genes.protein.regulatio

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樓主: fitful
21#
發(fā)表于 2025-3-25 06:53:20 | 只看該作者
22#
發(fā)表于 2025-3-25 08:41:09 | 只看該作者
Rb and Cellular Differentiation, both tumor formation and apoptosis are three cardinal features of terminal differentiation. Upon functional or genetic inactivation of pRb, cells from skeletal muscle, neuronal and hematopoi-etic lineages exhibit higher extent of apoptosis, fail to permanently exit the cell cycle and showincomplete differentiation.
23#
發(fā)表于 2025-3-25 13:40:53 | 只看該作者
ptosis and cancer.Includes supplementary material: .Rb and Tumorigenesis examines how recent advances have demonstrated the interaction of Rb with chromatin remodeling enzymes. This new title explores the potential roles of these interactions in Rb functions and provides some evidence that distinct
24#
發(fā)表于 2025-3-25 16:25:43 | 只看該作者
25#
發(fā)表于 2025-3-25 23:26:24 | 只看該作者
Regulation of E2F-Responsive Genes through Histone Modifications,l of cell growth. Many recent results have indicated that Rb represses transcription through proteins acting on chromatin structure. The purpose of this chapter is to review these results, and to discuss the possible mechanisms by which accurate regulation of E2F-responsive genes is achieved.
26#
發(fā)表于 2025-3-26 01:53:54 | 只看該作者
27#
發(fā)表于 2025-3-26 07:46:07 | 只看該作者
omatin remodeling enzymes. This new title explores the potential roles of these interactions in Rb functions and provides some evidence that distinct Rb co-repressor may target different genes in different phases of the cell cycle. .978-1-4899-9442-4978-0-387-33915-3
28#
發(fā)表于 2025-3-26 10:40:41 | 只看該作者
29#
發(fā)表于 2025-3-26 13:31:43 | 只看該作者
Medical Intelligence Unithttp://image.papertrans.cn/r/image/821691.jpg
30#
發(fā)表于 2025-3-26 18:27:18 | 只看該作者
Book 2006.Rb and Tumorigenesis examines how recent advances have demonstrated the interaction of Rb with chromatin remodeling enzymes. This new title explores the potential roles of these interactions in Rb functions and provides some evidence that distinct Rb co-repressor may target different genes in different phases of the cell cycle. .
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