Titlebook: Radiation Toxicity: A Practical Medical Guide; William Small,Gayle E. Woloschak Book 20061st edition Springer-Verlag US 2006 Tumor.brain.b

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MOAT

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Introduction,f tumors for both curative and palliative indications. Advances in treatment delivery, diagnostic imaging, and treatment planning systems have improved tumor control and, in many cases, reduced toxicity.

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The Management of Radiation-Induced Brain Injury,tegies, including observation, surgery, radiation therapy, and/or chemotherapy, are employed. Shown in Table 1 are the usual radiation doses used to treat primary and metastatic brain and spinal cord tumors, which span a broad range of total doses and doses per fraction. Regardless of the type of CN

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Rankle

Radiation Pneumonitis and Esophagitis in Thoracic Irradiation,ue complications limit our ability to administer the dose necessary for tumor control. Tumor control probability (TCP) for a given tumor is represented by a sigmoid curve in which an increase in dose results in greater tumor cell kill. Likewise, the normal tissue complication probability (NTCP) is r

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Upper Gastrointestinal Tract, often limits the radiation doses that can be given. Radiation therapy causes both acute and late effects. The acute effects of radiation include mucosal denudation, while late effects consist of fibrotic changes leading to decreased mobility and ischemia. Multifield and conformal radiation therapy,

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