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Titlebook: Programmed Cell Death in Cancer Progression and Therapy; Roya Khosravi-Far,Eileen White Book 2008 The Editor(s) (if applicable) and The Au

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書目名稱Programmed Cell Death in Cancer Progression and Therapy
編輯Roya Khosravi-Far,Eileen White
視頻videohttp://file.papertrans.cn/760/759863/759863.mp4
概述Up-to-date and comprehensive description of Molecular Mechanism of PCD in Cancer Progression and Therapy.Up-to-date and comprehensive information regarding the current therapeutic strategies that util
叢書名稱Advances in Experimental Medicine and Biology
圖書封面Titlebook: Programmed Cell Death in Cancer Progression and Therapy;  Roya Khosravi-Far,Eileen White Book 2008 The Editor(s) (if applicable) and The Au
描述.Programmed cell death (PCD) plays pivotal roles in tumor progression, cancer therapeutics and resistance of tumor cells to therapy. With the discovery of key mechanisms that are involved in mediating PCD and in promoting resistance to therapy, design of therapeutic approaches for promoting tumor-selective cell death has risen dramatically. With this book, we give a comprehensive overview of the mechanisms that are involved in mediating and regulating PCD in cancer. We also provide a detailed indication of the utility of PCD in cancer therapy. This book will be a valuable resource for researchers investigating the role of PCD in cancer and other diseases, researchers investigating the molecular mechanism of chemotherapeutic agents and drug-resistance in cancer and for physicians using chemotherapeutic agents. Additionally, this book will be a important educational source for PhD students specializing in cell biology, immunology and MD students interested in Oncology and Cancer Therapeutics..
出版日期Book 2008
關鍵詞Cell death, apoptosis, cancer research,Khosravi-Far; apoptosis; cell biology; regulation; tumorigenesis
版次1
doihttps://doi.org/10.1007/978-1-4020-6554-5
isbn_softcover978-90-481-7668-7
isbn_ebook978-1-4020-6554-5Series ISSN 0065-2598 Series E-ISSN 2214-8019
issn_series 0065-2598
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature B.V. 200
The information of publication is updating

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