Titlebook: Oxygen Transport to Tissue XL; Oliver Thews,Joseph C. LaManna,David K. Harrison Book 2018 Springer International Publishing AG, part of Sp

macular-edema

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Impact of Aging on Metabolic Changes in the Ketotic Rat Brain: Glucose, Oxidative and 4-HNE Metaboli inflammatory mediators, and increased protective pathways. Oxidative injury to cells is often associated with lipid peroxidation. Accumulation of intermediary products of lipid peroxidation includes 4-hydroxynonenal (HNE; a toxic lipid peroxidation intermediate). We investigated the metabolic effec

DAFT

Increases in Microvascular Perfusion and Tissue Oxygenation via Vasodilatation After Anodal Transcra current stimulation (tDCS) has emerged as a promising electroceutical therapeutic intervention possibly suitable for TBI; however, due to limited animal studies the mechanisms and optimal parameters are unknown. Using a mouse model of TBI we evaluated the acute effects of the anodal tDCS on cerebra

Pathogen

Meditative

Indent

Comparison of Quantitative and Qualitative Oxygen Extraction Fraction (OEF) in Acute Stroke Patientsatients with acute stroke and large vessel occlusive vascular disease. Early reports showed low morbidity and good outcomes. However, a large clinical trial in 1985 reported a failure of extracranial-intracranial (EC/IC) bypass to show benefit in reducing the risk of stroke compared to best medical

quiet-sleep

grieve

Acquired

Differences in Tissue Oxygenation, Perfusion and Optical Properties in Brain Areas Affected by Strokain disorders including cerebrovascular diseases. However, it is not known whether NIRS allow us to measure CBO correctly in patients with abnormal cortices where optical characteristics such as optical pathlength (OP) may differ from those in normal cortex. In the present study, employing a time-re

怒目而視

Changes of Doublecortin-Immunoreactive Cells from the Acute Phase to Chronic Phase After Transient Ger transient global brain ischemia (GBI) in the cingulate cortex (Cg) of rats. In the present study, we examined the changes of DCX-ir cells from the acute to the chronic phase after GBI in rats. Transient GBI was induced by a four-vessel occlusion model as described previously. Thirty-six rats were