Titlebook: Oxidative Stress in Skeletal Muscle; A. Z. Reznick (Senior Lecturer and Researcher),L. Book 1998 Springer Basel AG 1998 Lipid.Oxidation.a

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Antioxidant enzyme response to exercise and training in the skeletal muscle,eption of strict anaerobes and a few species of bacteria, most aerobic organisms are equipped with a host of enzymes that are directly or indirectly involved in the antioxidant defence against ROS. Enzymes that provide primary defences include superoxide dismutase (SOD), catalase (CAT), and glutathi

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Glutathione: A key role in skeletal muscle metabolism,H-dependent enzymes and remarkable GSH synthesizing ability of the skeletal muscle suggest that this tissue is a significant component of the complex inter-organ GSH homoeostasis. The hypothesis that skeletal muscle is a major player in whole body GSH metabolism has also been strongly supported by s

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Oxidative stress and Ca2+ transport in skeletal and cardiac sarcoplasmic reticulum,mpairment of membrane Ca. transport results in tissue injury (Rasmussen and Barrett, 1984; Thomas et al., 1996). In striated muscle, the signal that triggers force generation is a rise in cytoplasmic free Ca.-concentration (Ca.), whereas the removal of Ca. imparts a state of relaxation. Based on sop

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Effect of growth hormone on oxidative stress in immobilized muscles of old animals,crease in bone and joint disorders and muscle weakness. With reduced muscle activity or limb immobilization, there is also a progressive involution of muscle tissue, which has been termed sarcopenia. It has been proposed that growth hormone (GH) could counteract some of these ageing changes observed

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The diaphragm and oxidative stress,mals. It is now clear that increased diaphragmatic contractile activity promotes the production of ROS. Further, experimental evidence demonstrates that in resting muscle, a basal level of ROS are essential for optimal regulation of E-C coupling. In contrast, synthesis and release of NO inhibits E-C

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