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Titlebook: Oxidative Stress in Heart Diseases; Sajal Chakraborti,Naranjan S Dhalla,Madhu Dikshit Book 2019 Springer Nature Singapore Pte Ltd. 2019 he

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發(fā)表于 2025-3-23 12:44:15 | 只看該作者
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發(fā)表于 2025-3-23 16:24:06 | 只看該作者
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發(fā)表于 2025-3-23 21:24:27 | 只看該作者
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發(fā)表于 2025-3-24 01:22:55 | 只看該作者
Aging and Cardiovascular Diseases: The Role of Cellular Senescencend atherosclerosis. Existence of senescent vascular cells indicates that cellular senescence has a causative role in the pathology of cardiovascular diseases. There is also accumulating evidence that impairment in systemic metabolism is connected by cellular senescence. Therefore,?clearance of senes
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發(fā)表于 2025-3-24 04:27:41 | 只看該作者
Influence of Genetic Factor on Oxidative Stress Mediated Heart Damagere formulated in order to control the oxidative heart injury. This paper refers to the previously stated principles and presents only a part of abundant current knowledge about the oxidative stress genetics in heart diseases.
16#
發(fā)表于 2025-3-24 06:38:27 | 只看該作者
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發(fā)表于 2025-3-24 11:47:24 | 只看該作者
Redox Aspects of Myocardial Ischemia/Reperfusion Injury and Cardioprotectiondeleterious role of ROS/RNS in the cardiovascular field are considered in the light of their effect as either conditioning agents or damaging agents in ischemia/reperfusion scenario. The role of nitric oxide, nitration and nitrosylation in cardioprotection and redox regulation of transcription in ca
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發(fā)表于 2025-3-24 16:26:58 | 只看該作者
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發(fā)表于 2025-3-24 20:54:23 | 只看該作者
Role of Oxidative Stress and Cardiovascular Risk Factors in Ischemic Heart Diseasee of atherosclerosis, thrombosis and hypertension. Accordingly, inhibition of ROS-producing enzymes as well as mitochondrial reverse electron transport can be seen to normalize endothelial vascular function and prevent the development of cardiovascular risk factors for IHD. This article is intended
20#
發(fā)表于 2025-3-25 00:52:41 | 只看該作者
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