Titlebook: Oxidative Stress and Aging; R. G. Cutler,L. Packer,A. Mori Conference proceedings 1995 Birkh?user Verlag, Basel, Switzerland 1995 Biochemi

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Cellular aging and oxidative stresscatalase activity and reduced glutathione concentration were decreased. The . lifespan of human embryonic fibroblasts is perturbed by oxidative stress. The lifespan was shortened by treatment with hyperbaric oxygen and depended on cellular reduced glutathione concentration.

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stry and its possible involvement in both normal essential biology and age- related disease and dysfunction. Much of this growth in the traditionally separate sciences of chemistry and molecular gerontology occurred independently, with little interaction or communi- cation between the scientists wor

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Mitochondria, free radicals, neurodegeneration and agingry chain function contribute to the aging process. This chapter will review the lessons that can be learned from our study of inborn metabolic defects of the respiratory chain in understanding the functional importance of cumulative mtDNA mutations. The potential role of mitochondrial toxins in human disease will also be discussed.

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