Titlebook: Oral Mucosal Immunity and Microbiome; Georgios N. Belibasakis,George Hajishengallis,Mich Conference proceedings 2019 Springer Nature Switz

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A Potential Role of Phospholipase 2 Group IIA (PLA,-IIA) in ,-Induced Oral Dysbiosis,t–microbe symbiotic relationship leading to oral dysbiosis remain unclear. We have recently demonstrated that . specifically increased the antimicrobial properties of oral epithelial cells, through a strong induction of the expression of PLA.-IIA in a mechanism that involves activation of the Notch-

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spp. in Experimental Models of Colitis,ed by the commensal microbiota to the host, including the synthesis of vitamins, the degradation of complex polysaccharides into small chain fatty acids (which are essential for the maintenance of the intestinal epithelial barrier), and, finally, the outcompetition of pathogens that accidentally gai

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T Helper 17 Cells as Pathogenic Drivers of Periodontitis,hese cells have been described as orchestrators of immunopathology in several human inflammatory conditions including psoriasis, rheumatoid arthritis, and inflammatory bowel disease. More recently, the crucial role of Th17 cells in the regulation of immunity and protection of barrier sites has been

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Neutrophil Interaction with Emerging Oral Pathogens: A Novel View of the Disease Paradigm,ression depends on the disruption of host–microbe homeostasis in the periodontal tissue. This disruption is marked by a shift in the composition of the polymicrobial oral community from a symbiotic to a dysbiotic, more complex community that is capable of evading killing while promoting inflammation

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Advances in Oral Mucosal Immunity and the Microbiome,bstantial thematic overlap among all sessions, which promoted constant involvement of the participating scientists. An important hallmark was the active debate between oral microbiologists and oral immunologists, who explored new ideas and potential research collaborations, a crucial aspect for brid