Titlebook: Oncogenes in B-Cell Neoplasia; Workshop at the Nati Michael Potter,Fritz Melchers,Martin Weigert Book 1984 The Editor(s) (if applicable) an

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J. J. Mond,C. Thompson,M. Schaefer,F. D. Finkelman,R. Robb

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Detection of an Adenovirus E1A-like Activity in Mammalian Cellset al., 1979; Nevins, 1981). We have previously proposed that the E1A gene product mediates transcriptional activation through an indirect mechanism, possibly involving the inactivation of a cellular negative acting regulatory element (Nevins, 1981). This notion has been strengthened by the findings

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Does the Large T Protein of Polyoma Virus Regulate the Expression of the Cellular myc Gene?t al., 1981), . mutant NG18 (Schaffhausen et al., 1978)), changes were observed in the behaviour in culture of rodent embryo fibroblast cells in primary cultures (REF) (Rassoulzadegan et al., 1982, 1983): the cells grew in culture for an unlimited number of generations (“immortalization”) and grew i

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Genome Structure and Host Range Restriction of the Lymphotropic Papovavirus (LPV): Identification of African Green Monkey B-lymphoblastoid line and subsequently adapted to the human B-lymphoma line BJA-B. LPV shows a highly restricted host range in vitro. Only continuously growing cells of primate B-lymphocyte origin, i.e. B-lymphoma lines and B-lymphoblastoid cell lines immortalised by Epstein Ba

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Structural and Functional Analysis of ras and Blym Oncogenesnes which are activated in human neoplasms. Activated oncogenes of the . family have been detected in approximately 20% of human neoplasms, including carcinomas, sarcomas, neuroblastomas, leukemia, and lymphomas (reviewed in Cooper and Lane, 1984). Thus, . genes can apparently contribute to the deve

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Structural Organization of Mouse c-myb Locus and the Mechanism of its Rearrangement in ABPL-2 Tumor MuLV) and cellular sequences present within the normal mouse genome. The latter sequences, termed ., appear to code for the transforming properties of the virus (for review see Rosenberg and Baltimore, 1980). This virus induces in adult BALB/c mice a variety of lymphoid neoplasms predominantly of th

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Follicular Hyperplasia in the Prelymphomatous Avian Bursa: Relationship to the Incidence of B-Cell Llymphomas first appear from 8 to 12 weeks after virus infection and eventually metastasize to the spleen, liver and kidneys, resulting in death of the animal (Purchase and Burmester, 1978). The tumor cells are characterized by both cytoplasmic and cell surface immunoglobulin of the IgM isotype (Coop

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Transferrin Receptors Regulate Proliferation of Normal and Malignant B Cellsgh serum transferrin concentrations remain consistently high (in the 4 mg/ml range), resting lymphocytes do not possess detectable transferrin receptors and do not respond to transferrin (Tormey et al. 1972; Dillner-Centerlind et al., 1979; Larrick and Cresswell, 1979), suggesting that induction and