Titlebook: Obesity and Lipotoxicity; Ayse Basak Engin,Atilla Engin Book 20171st edition The Editor(s) (if applicable) and The Author(s), under exclus

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Endothelial Dysfunction in Obesity, cells, the balance between NO synthesis and ROS, insulin signaling and NO production, and decrease in .-arginine/endogenous asymmetric dimethyl-.-arginine (ADMA) ratio are discussed in connection with endothelial dysfunction in obesity.

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Influence of Antioxidants on Leptin Metabolism and its Role in the Pathogenesis of Obesity,nsufficient burning of calories but there is also a link to the regulation of food intake behavior. If the . findings can be extrapolated to the . situation, consumption of antioxidant supplemented food could lead to decreased leptin release and contribute to an obesogenic environment. This aspect s

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Non-Alcoholic Fatty Liver Disease,s, autophagy, and inflammation induce NASH progression. In the “first hit” the hepatic concentrations of diacylglycerol increase with rising saturated liver fat content in human NAFLD. Activities of mitochondrial respiratory chain complexes are decreased in liver tissue of patients with NASH. Furthe

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t. Ein guter Stumpf mit guter Prothese ist sehr oft besser als eine funktionsschwache, dauernd Schmerzen bereitende Gliedma?e. Auch im neuesten amerikanischen Schrifttum wird sehr eindringlich von den Grenzen der Replantation gesprochen. Wir sollten uns hier durch sensationelle Illustriertenartikel