Titlebook: Obesity; The Medical Practiti Robin P. Blackstone Book 2016 Springer International Publishing Switzerland 2016 Energy Metabolism.Reflux.Sle

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Mammal

祖先

Epidemiology, Measurement, and Cost of Obesity,emic is fueled by the growing rate of obesity in adolescents of 17?%. Healthcare systems have the responsibility to provide care to this burgeoning group of people. Accurate measurement and tracking of a patient’s BMI is critical. As a screening tool it may identify patients with a BMI of 25?kg/m. a

Binge-Drinking

Prejudice, Discrimination, and the Preferred Approach to the Patient with Obesity,are workers. In the healthcare environment, bias leads to inconsistent evaluation and management of obesity, discourages the patient from seeking care, and increases the cost of care. To eliminate bias, practitioners and staff must first identify their personal attitudes and misinformed beliefs abou

Hectic

欲望

The Biology of Adipose Tissue,the development of obesity fat becomes sick, causing adipose tissue to dysfunction. Adipose tissue dysfunction is the key tipping point in metabolic disease. Adipose tissue dysfunction is the final common pathway of inflammation and?it induces insulin resistance locally (adipose) and distally (muscl

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Obesity-Related Diseases and Syndromes: Insulin Resistance, Type 2 Diabetes Mellitus, Non-alcoholicose tissue becomes “sick.” Adisopathy or “sick fat” leads to a host of other obesity-related diseases. In this chapter the development of adisopathy is discussed in detail with an emphasis on insulin resistance and the role of mitochondria, hormones, inflammatory markers and leptin. This chapter des

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STANT

Pediatric Obesity,y 2020. Rates of childhood and adolescent obesity are increasing globally, as well. We blame the parents, the schools, the environment, computers, and cell phones – the list is endless. The question is: what will it take to reverse the trend? This chapter explains in detail why epigenetics and fetal

Agility

Fundamentals of Diet, Exercise, and Behavior Modification, is far easier to keep from gaining weight than to lose it. As explained in previous chapters excessive weight gain causes epigenetic changes that modulate the genes that control metabolism, hunger and satiety. These changes become hard wired in the individual with obesity, thereby"genetically reset