Titlebook: Nutrition and Heart Disease; Herbert K. Naito Book 1982 Spectrum Publications, Inc. 1982 heart.heart disease

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Quantitation of Lesions During Progression and Regression of Atherosclerosis in Rhesus Monkeys,iet, either alone or in combination with cholestyramine, will lead to regression of advanced atherosclerosis (Wissler et al., 1975; Vesselinovitch et al., 1976; Wissler and Vesselinovitch, 1976; Vesselinovitch et al., 1977; Wissler and Vesselinovitch, 1977; Vesselinovitch et al., 1978). Current stud

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Atherosclerosis and Diet, The Framingham, Massachusetts study (Kagan et al., 1962) established three major risk factors: (1) elevated serum cholesterol levels (above 240 mg/dl); (2) elevated systolic blood pressure (above 160 mm Hg); and (3) excessive cigarette smoking (over 20 cigarettes per day). A recent comparison of AS

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Systematic Approach to the Treatment of Hereditary Hyperlipidemia,isk factor for coronary heart disease. The physician is thus justified in making an all-out effort to reduce elevated plasma lipid levels in the hope of preventing, arresting, or reverting the atherosclerotic process. Indeed, there is considerable evidence in experimental animals (Armstrong, 1976) a

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Nutritional Modification for Prevention and Treatment of Hyperlipidemia and Hyperlipoproteinemia,lerosis, we are still not sure how the initiating and accelerating factors associated with this degenerative disease process interact and manifest themselves. One reason is that the disease process itself is complex, probably developing through a multitude of processes. Early in 1954, Dr. Page intro

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Serum Lipids in Total Parenteral Nutrition,of patients when their gastrointestinal system does not adequately function to provide for these needs. There are a number of disease states in which the gastrointestinal tract is not functional in absorbing sufficient nutrients to provide for adequate nutrition. Some of these conditions include obs

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