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Titlebook: Normal Testicular Descent and the Aetiology of Cryptorchidism; John M. Hutson,Masaru Terada,Martyn P. L. Williams Book 1996 The Editor(s)

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發(fā)表于 2025-3-21 19:51:41 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Normal Testicular Descent and the Aetiology of Cryptorchidism
編輯John M. Hutson,Masaru Terada,Martyn P. L. Williams
視頻videohttp://file.papertrans.cn/669/668034/668034.mp4
叢書名稱Advances in Anatomy, Embryology and Cell Biology
圖書封面Titlebook: Normal Testicular Descent and the Aetiology of Cryptorchidism;  John M. Hutson,Masaru Terada,Martyn P. L. Williams Book 1996 The Editor(s)
描述This review describes recent research into the cause of testicular descent and discusses two controversial theories on the mechanism. New animal models of cryptorchidism are described which shed light on the possible aetiology. A brief summary is given of current management, highlighting the controversies of timing of surgery, use of hormone therapy and congenital versus ‘a(chǎn)cquired‘ undescended testes. New information about germ cell development in the postnatal testis is changing the way we view treatment and the ultimate prognosis.
出版日期Book 1996
關(guān)鍵詞Cryptorchidism; Hodenhochstand; Krypptorchismus; Testicular Descent; cell; development; hormone; hormone th
版次1
doihttps://doi.org/10.1007/978-3-642-61026-4
isbn_softcover978-3-540-60283-5
isbn_ebook978-3-642-61026-4Series ISSN 0301-5556 Series E-ISSN 2192-7065
issn_series 0301-5556
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
The information of publication is updating

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沙發(fā)
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地板
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發(fā)表于 2025-3-22 14:50:10 | 只看該作者
John M. Hutson,Masaru Terada,Baiyun Zhou,Martyn P. L. Williams
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發(fā)表于 2025-3-22 19:53:26 | 只看該作者
John M. Hutson,Masaru Terada,Baiyun Zhou,Martyn P. L. Williams
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Conclusions,n cryptorchid rodents, suggesting that it may be an important factor in many species. This has led to speculation that CGRP treatment may be used therapeutically in causing descent of undescended testes. Investigation of germ cell development in the immature mouse has revealed a putative role for MI
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Normal Testicular Descent and the Aetiology of Cryptorchidism
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