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Titlebook: Non-Neuronal Mechanisms of Brain Damage and Repair After Stroke; Jun Chen,John H. Zhang,Xiaoming Hu Book 2016 Springer International Publi

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31#
發(fā)表于 2025-3-26 23:27:04 | 只看該作者
Angiogenesis: A Realistic Therapy for Ischemic Strokenal cerebral blood supply, which is very critical for the stroke outcomes and post-stroke functional recovery both in human and experimental animal models. In this review, we summarize current knowledge about angiogenesis processes following ischemic stroke and potential benefit of future developmen
32#
發(fā)表于 2025-3-27 03:26:30 | 只看該作者
33#
發(fā)表于 2025-3-27 07:51:53 | 只看該作者
Crosstalk Between Cerebral Endothelium and Oligodendrocyte After Strokel interactions are not static—the trophic coupling is disturbed during the acute phase after brain injury, with the potential to recover in the chronic phase to promote brain remodeling and repairing. Oligodendrocyte lineage cells play critical roles in white matter function. Therefore, a deeper und
34#
發(fā)表于 2025-3-27 11:51:48 | 只看該作者
The Peripheral Immune Response to Strokeshow that allogeneic skin grafts transplanted into the anterior chamber of the eye or the brain of rabbits were efficiently rejected when the host was previously immunized with donor-derived cells and that this rejection was dependent on vascularization of the graft. The resistance of the CNS to mou
35#
發(fā)表于 2025-3-27 17:09:00 | 只看該作者
36#
發(fā)表于 2025-3-27 18:04:24 | 只看該作者
B-Cells in Stroke and Preconditioning-Induced Protection Against Strokell be summarized that highlights the role for B-cells in acute CNS neuroprotection after stroke, as well as new data suggesting that B-cells may be detrimental to long-term cognitive function in experimental stroke. Finally, this chapter will focus on preconditioning, a method by which non-injurious
37#
發(fā)表于 2025-3-27 22:26:32 | 只看該作者
Mast Cell as an Early Responder in Ischemic Brain Injuryeterious role following acute cerebral ischemia. Inhibition of MC effects by pharmacological agents or gene manipulation has been demonstrated to alleviate postischemic blood–brain barrier (BBB) failure, expansive edema, inflammation, neuroinjury, and neurologic dysfunction in models of experimental
38#
發(fā)表于 2025-3-28 04:08:18 | 只看該作者
Roles of Neutrophils in Strokehils has resulted in improved behavioral outcomes and reduced infarct volumes. However, early human studies including the anti-ICAM-1 trial have not been successful. Potentially a more neutrophil specific strategy may be required. Neutrophil has beneficial properties which may be important to preser
39#
發(fā)表于 2025-3-28 06:46:19 | 只看該作者
The Function of Cytokines in Ischemic Stroketiplicity of roles, and the various components of cerebral infarct expansion, much work is needed to decipher which key signals should be targeted. In addition to identifying putative pro- and anti-inflammatory cytokines for therapeutic interventions, the timing of intervention will likely be critic
40#
發(fā)表于 2025-3-28 12:57:01 | 只看該作者
Structural Alterations to the Endothelial Tight Junction Complex During Strokeerlying mechanisms and potential therapeutic interventions. While the effects of stroke on the BBB have long been considered as secondary to neuronal injury, increasing evidence stresses the importance of neurovascular injury in stroke and that BBB damage may be a cause rather than a result of stroke-induced parenchymal damage.
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