Titlebook: Non-Alcoholic Steatohepatitis; Methods and Protocol Devanand Sarkar Book 2022 The Editor(s) (if applicable) and The Author(s), under exclus

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Analysis of Liver Responses to Non-alcoholic Steatohepatitis by mRNA-Sequencing,hough NASH is associated with excessive liver lipid accumulation, hepatocyte injury, inflammation, and fibrosis, its etiology remains incompletely understood. These can be characterized by determining transcriptional changes in specific genes previously found to be involved in these processes. As an

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Single Cell RNA Sequencing in NASH,ohepatitis (NASH), scRNA-seq is particularly powerful to understand non-parenchymal cell heterogeneity in the liver, e.g. for inflammatory cells. Myeloid immune cells, particularly macrophages, play a critical role in response of the innate immune system and significantly contribute to the progressi

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Computational Pipeline for Next-Generation Sequencing (NGS) Studies in Genetics of NASH,fatty liver disease (NAFLD). Genome-wide association studies, whole exome sequencing, and sequencing of entire genes are used to identify variants and/or mutations that predispose to the disease pathogenesis. Here, we present a tutorial that may guide readers to manage high volume of genetics data i

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Metabolic Phenotyping in Mice with NASH Using Indirect Calorimetry,etes, non-alcoholic steatohepatitis (NASH), cardiovascular diseases, and cancer. Metabolic phenotyping is a set of techniques for studying metabolic dysfunction and behavior information including energy expenditure, body weight gain, glucose homeostasis, and lipid profile. Among different metabolic

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Assessment of Lipotoxic Endoplasmic Reticulum (ER) Stress in Nonalcoholic Steatohepatitis (NASH),asmic reticulum (ER) stress. Consequently, the unfolded protein response (UPR) is initiated, driven by three key ER transmembrane proteins, resulting in downstream responses that are dynamic and interconnected. Thus, careful interrogation of these pathways is required to investigate the complex role

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Methods in Molecular Biologyhttp://image.papertrans.cn/n/image/666843.jpg

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https://doi.org/10.1007/978-1-0716-2128-8CRISPR; Kupffer cells; Inflammasome; cell death; NASH

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