Titlebook: Nitric Oxide, Cytochromes P450, and Sexual Steroid Hormones; Jack R. Lancaster,John F. Parkinson Conference proceedings 1997 Springer-Verl

Blood-Vessels

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Regulation of Uterine and Cervical Function by Nitric Oxide,ds with delivery of the fetus and placenta. It is a procedure that involves progession of uterine contractility from inactive to vigorously active and softening and opening of the rigid and closed cervix. These crucial steps are thought to be regulated by the coordinated change in various systems in

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,Nitric Oxide in Arthritis: It’s Probably There But What’s It Doing?,s occur within the joints and all forms of arthritis produce articular inflammation or cartilage destruction or both. Patients with osteoarthritis (OA), for example, suffer considerable loss of cartilage with only low levels of inflammation, whereas the joints of patients with systemic lupus erythem

TOXIN

Nitric Oxide and the Skeleton,se: (1) it inhibits preosteoclast proliferation; (2) a “puff” of NO, triggered by calcium-stimulated constitutive nitric oxide synthase (cNOS) facilitates cell detachment and subsequent movement; (3) the radical is probably involved in the final apoptotic phase of the cycle; (4) the gas radical play

Neutral-Spine

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Estrogen, Nitric Oxide, and Primate Atherosclerosis,reduced by about 50% in postmenopausal women who take conjugated equine estrogens (CEE) daily (Bush 1990; Stampfer and Colditz 1991). Observational studies of postmenopausal women undergoing angiography for chest pain have shown that coronary artery occlusions are fewer among estrogen users than non

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Nitric Oxide Deficiency in Preeclampsia,ng cause of infant morbidity in obstetrics after preterm birth. Moreover, preeclampsia is the largest single cause of maternal mortality in developed countries. The etiology of this disease is largely unknown and a causative therapy is yet not available. For many years preeclampsia was thought to re

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Endothelial Cells, Estrogen and Angiogenesis,rt attacks and strokes compared with men. After menopause, the incidence of atherosclerotic cardiovascular disease rapidly approximates that of men (Stampfer et al. 1990). Moreover, several studies have indicated that postmenopausal estrogen replacement therapy may decrease the incidence of subseque

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