Titlebook: Nitric Oxide and Free Radicals in Peripheral Neurotransmission; Stanley Kalsner Book 2000 Springer Science+Business Media New York 2000 Ne

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Presynaptic Modulation of Peripheral Nitrergic Neurotransmission, noradrenaline, acetylcholine, and dopamine has developed (.,.,.). In both the central and peripheral nervous system, the release of these neurotransmitters can, indeed, be regulated via presynaptic receptors on the axon terminals. Modulation can be exerted by the neuron’s own neurotransmitter via p

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Neural Nitric Oxide Synthase and Neutrophil Function, synthase converts L-arginine to nitric oxide (NO) and L-citrulline utilizing the electrons generated by the reduction of oxygen to superoxide, hydrogen peroxide, and ultimately water (.,.; .). At least three isozymes of NOS exist, each of which is derived from a different gene (.). Endothelial NOS

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Nitric Oxide Toxicity in Neuronal Injury and Degeneration,has a role in the induction of motor neuron death during development, after injury, and in pathological conditions such as amyotrophic lateral sclerosis (ALS). We also speculate about the mechanisms of motor neuron death induced by ALS mutant superoxide dismutases (SOD).

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Peroxynitrite and Nitrergic Neural Transmission: Pathophysiological Implications,xide anion radical (O.’1; superoxide can be converted spontaneously or by the catalytic superoxide dismutase (SOD) to the nonradical molecule hydrogen peroxide (H.0.), which can be toxic at high concentrations and, more importantly, can be reduced to the hydroxyl radical (’OH) (.). Superoxide is rel

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濕潤(rùn)

https://doi.org/10.1007/978-1-4612-1328-4Nervous System; blood vessel; endothelium; neurophysiology; neuroscience; nitric oxide; physiology; tissue