Titlebook: Neuroimmune Diseases; From Cells to the Li Hiroshi Mitoma,Mario Manto Living reference work 00th edition neuroimmunology.immunotherapy.aut

開(kāi)始從未

Stanislas Lagarde,Nathalie Villeneuve,Fabrice Bartolomeiwe need only look at the powers which he received from the Catholic Kings, Ferdinand and Isabella (1474–1504/16), to realise how indebted the founders of empire also were to those who, over the previous centuries, had elaborated institutions which were capable of being adapted to new challenges and

OMIT

Anti-IgLON5 Disease: A Cross Talk between Autoimmunity and Tauopathy,s the presence of antibodies against IgLON5, the fifth member of the IgLON family that belongs to the immunoglobulin superfamily of cell adhesion molecules predominantly expressed in the neuronal membrane. Patients with anti-IgLON5 disease are usually 60 years or older and develop a chronic course,

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溫室

Autoimmune Ataxias,cing (WGS), has also revealed that a substantial number of sporadic ataxias are not due to genetic defects but are likely to be immune-mediated for a proportion of them. At the same time, the recent identification of an increasing number of antibodies associated with sporadic ataxias has clarified t

Sputum

GNAW

Central Nervous System Vasculitis and Related Diseases,ther a manifestation of systemic vasculitis or a complication associated with certain specific pathologies, such as infections (viral, bacterial, or fungal), neoplasia, drugs, connective tissue diseases (e.g., systemic lupus erythematous, rheumatoid arthritis, and Sj?gren syndrome), and sarcoidosis.

運(yùn)氣

Chronic Inflammatory Demyelinating Polyneuropathy,fic diagnostic test, CIDP is diagnosed on the basis of the clinical presentation and demonstration of demyelination by electrodiagnostic or nerve biopsy studies. There are many phenotype variants of CIDP. Typical CIDP involves motor and sensory nerve dysfunction, with motor deficits reported in up t

Albinism

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Genetic Factors in Neuroimmune Diseases,or the peripheral nervous system. Unlike monogenic diseases, neuroimmune disorders do not follow Mendelian patterns of inheritance, and their genetic basis has been elusive for decades. It has been only recently that novel methodologies of analysis, such as the genome-wide association study (GWAS) p

Irksome

GFAP Autoimmune Astrocytopathy: Clinical and Immunological Characteristics,NS) associated with autoantibodies targeting GFAP. It presents as acute to subacute meningoencephalitis, affecting various CNS regions. The condition primarily affects individuals over 40?years old and shows a slight female predominance. Symptoms include fever, headache, encephalopathy, and ataxia,