Titlebook: Neurobiology of Essential Fatty Acids; Nicolas G. Bazan,Mary G. Murphy,Gino Toffano Book 1992 The Editor(s) (if applicable) and The Author

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https://doi.org/10.1007/978-1-4615-3426-6GABA; Glutamat; behavior; biology; brain; kinetics; lipide; neurobiology; pathophysiology; physiology; recepto

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0065-2598 membranes of neural tissue, are actually major determinants of the functional properties of neural cells and are intimately involved in brain disease processes. The two families of PUF A, which are derived from the essential polyenes, linoleic (18:26) and a-linolenic (18:33) acids, constitute a majo

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Modulation of Arachidonic Acid Metabolism In Cultured Rat Astroglial Cells By Long-Chain N-3 Fatty Aoducing and releasing molecules, such as the eicosanoids and other compounds derived from phospholipid hydrolysis, which are typical mediators of inflammation. Eicosanoids are generated in brain under various pathologic conditions, such as trauma, ischemia and convulsions, following the release of arachidonic acid (AA) from cell phospholipids.

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Extracellular Phospholipase A2 of a phospholipase, either directly or through mediators. Presumably this phospholipase is membrane associated or becomes so after translocation (Channon and Leslie, 1990). Because the bulk of cellular arachidonic acid is found esterified in the .position of membrane phospholipids, a phospholipase A. is the simplest and most logical enzyme