Titlebook: Neurobiology of Essential Fatty Acids; Nicolas G. Bazan,Mary G. Murphy,Gino Toffano Book 1992 The Editor(s) (if applicable) and The Author

AMOR

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偏狂癥

謙卑

Extracellular Phospholipase A2oduction of the prostaglandins, thromboxanes, and prostacyclins via the cyclooxygenase pathway, as well as the production of the leukotrienes and lipoxins via the lipoxygenase pathway, all depend on the availability of free arachidonic acids. As illustrated in Figure 1, control of the production of

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帽子

Reciprocal Regulation of Fatty Acid Release In The Brain By Gaba and Glutamatehe activity of protein kinase C, and changing ion channel conductance in the cell membrane. However, the neurotransmitters and other factors that control the release of FFA in neurons in normal or pathological states are not well defined. The following studies investigate the regulation of FFA relea

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NMDA Receptor-Mediated Arachidonic Acid Release In Neurons: Role In Signal Transduction and Patholog memory formation, in the induction of long-term potentiation, and also in seizure activity and neurotoxicity. In primary cultures of cerebellar neurons, agonists of NMDA receptors induce a dose-dependent release of [.H]arachidonic acid ([.H]AA), which is potentiated by activation of the glycine-pos

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刺耳

Modulation of Arachidonic Acid Metabolism In Cultured Rat Astroglial Cells By Long-Chain N-3 Fatty A types of neuromediators. Glial cells are also involved in processes which are activated by brain injury, and participate in inflammatory events by producing and releasing molecules, such as the eicosanoids and other compounds derived from phospholipid hydrolysis, which are typical mediators of infl