Titlebook: Nephrotoxicity in the experimental and clinical situation; Part 1 P. H. Bach,E. A. Lock Book 1987 Martinus Nijhoff Publishers 1987 Fixation

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E. A. Lockolute via hydrogen bonds and/or electrostatic forces related to their high dipolar momentum. At room temperature, the correlation times t of rotational and translational movements, of the order of 10. to 10. s in pure water may increase to 10. or even 10. in the hydration layer of macromolecules (Pa

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toAb reflect antigenic disturbances between endogenous B(a)P ligand and its cytosolic receptor(s), and disturbances in Ptdlns turn over. In the same time, a chemically-induced malignant tumor model was obtained after subcutaneously (s.c.) administration of a single dose of B(a)P (0.1mg/g body weight

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Correlating Structural and Functional Changes in Nephrotoxic Renal Injury, tubular injury in clinical forms of acute renal failure is not prominent. Hence, the question of whether morphological injury precedes or contributes to the development of renal dysfunction remains controversial, at least in the clinical setting.

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Assessment of the Kidney in Relation to Blood Pressure Regulation, in blood pressure. The “renal blood volume pressure regulatory system” or “the sodium homeostatic mechanism” fulfils such a role.. However the corollary of this is that renal dysfunction is the primary cause of hypertension, and this is one of the most enduring controversies of modern day pathophys