Titlebook: Nephrology; Volume I Michinobu Hatano (Professor of Medicine, Director) Book 1991 Springer Japan 1991 internal medicine.kidney.medicine.nep

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Pathogenesis of Glomerulonephritis — 1990nterstitial mononuclear cell infiltration to the progression of renal injury and declining function in glomerulonephritis is discussed. Insights gained from these developing areas of nephrologic research suggest important new approaches to the understanding and improved therapy of glomerulonephritis.

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ansformed cells are anchorage independent, grow in semisolid media to high saturation density, and show a disruption of the cytoskeleton, all of which are changes observed with other transformed cells and transforming genes. Papkoff et al. (9) demonstrated that cells acutely infected with Mo-MSV exp

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C. Craig Tisher,Kirsten M. Madsen,Jill W. Verlanderellular matrix interactions, such as tissue organization and development, glycoprotein catabolism, inflammation, fertilization and microbial attachment, are mediated, at least in part, by lectins that are present on the cell surface as integral or adsorbed components for both vertebrate and inverteb

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Masashi Imai,Koji Yoshitomi,Yoshiaki Kondo,Junichi Taniguchi,Chizuko Koseki,Taisuke Isozaki,Kaoru Taas helping to organize the cytoskeleton. This broad repertoire of responses is consistent with the current view that integrins are central mediators of information transfer between cells and the extracellular matrix, and sometimes other cells, both at the physical/structural level and at the level o

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Mechanisms of Na+, Cl?, and HCO 3 ? Transport in the Proximal Tubuletegrate these subcellular mechanisms into the overall functioning of the intact proximal tubule will be presented. Literature citations will be representative, rather than comprehensive, due to the time/space limitations imposed on this lecture/chapter.