| 書目名稱 | Molecular Mechanisms of Cancer | | 編輯 | Georg F. Weber | | 視頻video | http://file.papertrans.cn/639/638351/638351.mp4 | | 概述 | Comprehensive text.Highly systematic structure.Explains mechanisms of molecular interactions.Description of functions ranges from basic cellular mechanisms to specific clinical syndromes.Covers common | | 圖書封面 |  | | 描述 | Cancer may constitute the most extensively studied functions constitute a second line of defense that disease entity of our time. Nevertheless, our com- protects against transforming defects in oncogenes prehension of the cellular and molecular pathology or tumor-suppressor genes and are here considered of malignant transformation is incomplete. In view as metasuppressor genes. Advances in the molecular of the diverse clinical presentations of various explanations of growth dysregulation, metastasis malignancies, doubts may be raised as to whether it formation, extension of life span, and loss of ma- is appropriate to refer to cancer as one group of dis- tenance of genomic and epigenetic integrity in ease states. The notion of malignant tumors as a cancer suggest models for their causal connection. pathologic and pathophysiologic class of conditions The mechanisms of growth control, senescence, and begs the question for defining criteria that charac- anchorage dependence are linked on the molecular terize all malignant growths, regardless of their tis- level (Chapter 8). In cells that are not fully differ- sue of origin. Toward this goal, the recognition that tiated, the overactiva | | 出版日期 | Textbook 2007 | | 關(guān)鍵詞 | DNA; Immune System; angiogenesis; cell division; genes; molecular mechanisms; regulation; senescence; tissue | | 版次 | 1 | | doi | https://doi.org/10.1007/978-1-4020-6016-8 | | isbn_softcover | 978-90-481-7505-5 | | isbn_ebook | 978-1-4020-6016-8 | | copyright | Springer Science+Business Media B.V. 2007 |
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